Liberate Yourself- Breathwork Ceremony with Crystal Bowls Sound Bath

6463 Bird Road,Miami FL 33155

05 July, 2022

Description

Liberation Breathing is a technique that helps release any energies stuck inside of the body that is ready to come out. It is a connected breath that oxygenates the body and has proven to have numerous benefits. Everyone has their own experience and walk away feeling so much better for letting go of whatever it is that they were holding inside and brings clarity of mind and rests the nervous system. Please feel free to bring family, partners and friends. Also, bring water, a notepad with pencil/pen, yoga mat, and a blanket This event is open for both men and women. Details to know and help determine if this is for you.. Conscious, Connected Breathwork results in certain specific physiological changes in the body and also can result in intense physical and emotional release. Please be aware that these sessions are safe, illuminating, transformative and deeply healing, and can sometimes cause the following: * Tingling * Buzzing * Vibrating * Trembling, shaking or other physical sensations, including spontaneous physical movement * Tetany (tightness in the hands or sometimes the jaw- we will guide you on how to work with it and manually release it during the live session) * Somatic (body) unwinding * Deep emotional experiencing, release and catharsis * Trauma release * Experiencing life force energy (prana) moving through your body * Unlocking repressed sensual/orgasmic/shakti energies * Ecstatic, kundalini awakenings (usually for more advanced journeyers) BREATHWORK CONTRAINDICATIONS Breathwork results in certain specific physiological changes in the body and also can result in intense physical and emotional release. As a precaution, the following conditions are contraindicated in certain breathwork practices: - Pregnancy (at any stage) - Severe PTSD or trauma - Actively using recreational drugs - Taking any medication that alters brain chemistry like anti-anxiety, anti-depressant, ADD, OCD medications, etc. - Prescription blood thinners or anti-clotting medications - Osteoporosis - Detached Retina - Glaucoma - Kidney disease - High Blood Pressure - Cardiovascular disease, including angina, previous heart attack, or any other cardiovascular issues - Strokes or TIAs - Seizures, history of seizures or other brain/neurological condition or disease - Diagnosis of aneurysm (of any kind) - Uncontrolled thyroid conditions - Diabetes (any type) - Severe Asthma— for mild to moderate asthma, we can discuss but you must bring your inhaler to the session - Epilepsy - Prior diagnosis of bipolar disorder, schizophrenia or previous psychiatric condition - Severe psychosomatic disorders - Recent surgery, stitches or injury - Hospitalization for any psychiatric condition or emotional crisis within the last 10 years - Any other medical, psychiatric or physical conditions which would impair or affect ability to engage in any activities that involve deep physical and/or emotional release - The mRNA COVID Inoculation: At this time, clients must wait a minimum of 60 days after their final dosage (including boosters) with NO SIDE EFFECTS to engage Breathwork. This decision has been made in consultation with our insurance company, health professionals, legal team and through our own deep contemplation. At this time, the health effects of the Inoculation are still too unknown and not covered by our liability. Thus, we regret to inform you at this time that the COVID Inoculation(s) is a contraindication to our Breathwork until one has safely received all doses with NO side effects for a minimum of 60 days or more. We apologize for any inconvenience this may cause and will reevaluate our policy when clinical trials are completed in 2023, and more data has been collected and documented. If you are on the contraindication list, you are not a candidate for the full breathwork technique, but we are happy to discuss modified, gentle breathwork with you for a later date. Please do not continue to book as session if you have any of the contraindications listed above. The path to healing is a way to find your center and awaken your shakti. The inner medicine that creates alchemy from within. The shifts are profound and help merge you with your higher self. Study shows how slow breathing induces tranquility Mark Krasnow and his colleagues have identified a tiny cluster of neurons that link breathing to relaxation, attention, excitement and anxiety. Steve Fisch Mar 30 2017 Stanford Medicine - News Center By Bruce Goldman Try it. Breathe slowly and smoothly. A pervasive sense of calm descends. Now breathe rapidly and frenetically. Tension mounts. Why? It’s a question that has never been answered by science, until now. In a new study, researchers at the Stanford University School of Medicine and their colleagues have identified a handful of nerve cells in the brainstem that connect breathing to states of mind. A paper describing the findings were published March 31 in Science. Mark Krasnow, MD, PhD, professor of biochemistry, is the senior author. The lead author is former Stanford graduate student Kevin Yackle, MD, PhD, now a faculty fellow at the University of California-San Francisco. Medical practitioners sometimes prescribe breathing-control exercises for people with stress disorders. Similarly, the practice of pranayama — controlling breath in order to shift one’s consciousness from an aroused or even frantic state to a more meditative one — is a core component of virtually all varieties of yoga. “This study is intriguing because it provides a cellular and molecular understanding of how that might work,” Krasnow said. Tiny cluster of neurons The tiny cluster of neurons linking respiration to relaxation, attention, excitement and anxiety is located deep in the brainstem. This cluster, located in an area Krasnow calls the pacemaker for breathing, was discovered in mice by study co-author Jack Feldman, PhD, a professor of neurobiology at UCLA, who published his findings in 1991. An equivalent structure has since been identified in humans. Stanford Neurosciences Institute The diagram depicts the pathway (in green) that directly connects the brain's breathing center to the arousal center and the rest of the brain. Courtesy of the Krasnow lab “The respiratory pacemaker has, in some respects, a tougher job than its counterpart in the heart,” said Krasnow, who is also a Howard Hughes Medical Institute investigator. “Unlike the heart’s one-dimensional, slow-to-fast continuum, there are many distinct types of breaths: regular, excited, sighing, yawning, gasping, sleeping, laughing, sobbing. We wondered if different subtypes of neurons within the respiratory control center might be in charge of generating these different types of breath.” On that hunch, Yackle searched through public databases to assemble a list of genes that are preferentially activated in the part of the mouse brainstem where the breathing-control center resides. This center’s technical term is the pre-Bötzinger complex, or preBötC. He pinpointed a number of such genes, allowing the investigators to identify more than 60 separate neuronal subtypes, physically differentiated from one another by their gene-activation signatures but comingling in the preBötC like well-stirred spaghetti strands. The scientists were able to use these genes, and the protein products for which they are recipes, as markers allowing them to zero in on the different neuronal subtypes. Knocking out neurons Now the scientists could systematically assess the role of each neuronal subpopulation in laboratory mice. With advanced technologies, they could selectively destroy any one of these neuronal subtypes — and only that subtype — based on its unique signature of active genes. Then they could observe how this particular subtype’s loss affected the animals’ breathing. In 2016, in collaboration with Feldman, they succeeded in isolating a subpopulation of neurons in the preBötC that explicitly controls one type of breathing: sighing. Knocking out these neurons eliminated sighing but left other modes of breathing unaffected. The discovery was published in Nature in 2016. Stanford Neurosciences Institute, Kevin Yackle Krasnow and Yackle then set out to discover the respiratory role of another subpopulation of about 175 preBötC neurons distinguished by their shared expression of two genetic markers called Cdh9 and Dbx1. They bioengineered mice in which they could wipe out, at will, the neurons bearing both of these markers. But once these rodents had their Cdh9/Dbx1 neurons eliminated, they seemed to take the loss in stride. Unlike their sigh-deprived brethren, there was no lacuna in these mice’s portfolio of breathing variations. “I was initially disappointed,” said Yackle. But a few days afterward, he noticed something: For mice, the animals were extraordinarily calm. “If you put them in a novel environment, which normally stimulates lots of sniffing and exploration,” Yackle said, “they would just sit around grooming themselves” — evidence of what passes for mellowness when you’re a mouse. Chilling out Further analysis showed that while these mice still displayed the full palette of breathing varieties from sighs to sniffs, the relative proportions of those varieties had changed. There were fewer fast “active” and faster “sniffing” breaths, and more slow breaths associated with chilling out. The investigators surmised that rather than regulating breathing, these neurons were spying on it instead and reporting their finding to another structure in the brainstem. This structure, the locus coeruleus, sends projections to practically every part of the brain and drives arousal: waking us from sleep, maintaining our alertness and, if excessive, triggering anxiety and distress. It’s known that neurons in the locus coeruleus exhibit rhythmic behavior whose timing is correlated with that of breathing. In a series of experiments, the Stanford researchers proved that the preBötC neurons that express Cadh9 and Dbx1 not only project to the locus coeruleus — a new finding — but activate its long-distance-projections, promoting brainwide arousal. “If something’s impairing or accelerating your breathing, you need to know right away,” said Krasnow. “These 175 neurons, which tell the rest of the brain what’s going on, are absolutely critical.” “The preBötC now appears to play a key role in the effects of breathing on arousal and emotion, such as seen during meditation,” said Feldman. “We’re hopeful that understanding this center’s function will lead to therapies for stress, depression and other negative emotions.” Other Stanford co-authors are John Huguenard, PhD, professor of neurology and neurological sciences; Liqun Luo, PhD, professor of biology and an HHMI investigator; former postdoctoral scholar Lindsay Schwarz, PhD; and graduate student Jordan Sorkin. A researcher at the Chicago Medical School also co-authored the study. Krasnow is also executive director of the Wall Center for Pulmonary Vascular Disease, a member of the Stanford’s Neurosciences Institute, Cardiovascular Institute, Cancer Institute and Bio-X. The study was funded by the National Institutes of Health (grants HL70029 and HL40959) and HHMI. Stanford’s Department of Biochemistry also supported the work.